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The motif for peptide binding to the insulin-dependent diabetes mellitus-associated class II MHC molecule I-A(g7) validated by phage display library
INTERNATIONAL IMMUNOLOGY
NKT lymphocyte ontogeny and function are impaired in low antibody-producerBiozzi mice: gene mapping in the interval-specific congenic strains raisedfor immunomodulatory genes
INTERNATIONAL IMMUNOLOGY
A structural framework for deciphering the link between I-A(g7) and autoimmune diabetes
SCIENCE
The I-A(g7) MHC class II molecule linked to murine diabetes is a promiscuous peptide binder
JOURNAL OF IMMUNOLOGY
Early Th1 response in unprimed nonobese diabetic mice to the tyrosine phosphatase-like insulinoma-associated protein 2, an autoantigen in type 1 diabetes
JOURNAL OF IMMUNOLOGY
Low antibody responsiveness is found to be associated with resistance to chemical skin tumorigenesis in several lines of Biozzi mice
CANCER LETTERS
The influence of environmental and genetic factors on the disease resistance of fish
AQUACULTURE
A peptide binding motif for I-E-g7, the MHC class II molecule that protects E alpha-transgenic nonobese diabetic mice from autoimmune diabetes
JOURNAL OF IMMUNOLOGY
EFFECT OF GENETIC-MODIFICATION OF ACUTE INFLAMMATORY RESPONSIVENESS ON TUMORIGENESIS IN THE MOUSE
Carcinogenesis
IDENTIFICATION OF 2 QUANTITATIVE TRAIT LOCI INVOLVED IN ANTIBODY-PRODUCTION ON MOUSE-CHROMOSOME-8
Immunogenetics
GENETIC-REGULATION OF DERMATOPHAGOIDES PTERONYSSINUS-SPECIFIC IGE RESPONSIVENESS - A GENOME-WIDE MULTIPOINT LINKAGE ANALYSIS IN FAMILIES RECRUITED THROUGH 2 ASTHMATIC SIBS
Journal of allergy and clinical immunology
GENETIC-CONTROL OF IMMUNE RESPONSIVENESS, AGING AND TUMOR-INCIDENCE
Mechanism of ageing and development
FUNCTIONAL DIFFERENCES IN THE SPECIFIC B-CELL COMPARTMENT IN HIGH OR LOW ANTIBODY RESPONDER MICE
Scandinavian journal of immunology
T-HELPER SUBSET INVOLVEMENT IN 2 HIGH ANTIBODY RESPONDER LINES OF MICE (BIOZZI MICE) - HI (SUSCEPTIBLE) AND HII (RESISTANT) TO COLLAGEN-INDUCED ARTHRITIS
European Journal of Immunology
MLS-1 AND MLS-2 SUPERANTIGENS DO NOT CONTROL SUSCEPTIBILITY TO COLLAGEN-INDUCED ARTHRITIS IN H-I AND H-II MICE
Immunology