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Phenotypic characterization of Lith genes that determine susceptibility tocholesterol cholelithiasis in inbred mice: soluble pronucleating proteins in gallbladder and hepatic biles
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Chromosomal organization of candidate genes involved in cholesterol gallstone formation: a murine gallstone map. (vol 120, pg 221, 2001)
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Chromosomal organization of candidate genes involved in cholesterol gallstone formation: A murine gallstone map
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Treatment options for vascular ectasias of the gastric antrum (watermelon stomach)
DEUTSCHE MEDIZINISCHE WOCHENSCHRIFT
Diagnosis for vascular ectasias of the gastric antrum (watermelon stomach)
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Central pontine myelinolysis and myocardial infarction following severe hypoglycemia
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Cholic acid aids absorption, biliary secretion, and phase transitions of cholesterol in murine cholelithogenesis
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Long-term survival after diagnosis of hepatic metastatic VIPoma - Report of two cases with disparate courses and review of therapeutic options
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No pathophysiologic relationship of soluble biliary proteins to cholesterol crystallization in human bile
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Phenotypic characterization of Lith genes that determine susceptibility tocholesterol cholelithiasis in inbred mice: pathophysiology of biliary lipid secretion
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Phenotypic characterization of Lith genes that determine susceptibility tocholesterol cholelithiasis in inbred mice: integrated activities of hepatic lipid regulatory enzymes
JOURNAL OF LIPID RESEARCH
LOCALIZATION OF THE ILEAL SODIUM-BILE SALT COTRANSPORTER GENE (SLC10A2) TO MOUSE-CHROMOSOME-8
Mammalian genome
THE GENE ENCODING THE MULTISPECIFIC ORGANIC ANION TRANSPORTER (CMOAT)OF THE HEPATOCYTE CANALICULAR MEMBRANE MAPS TO MOUSE-CHROMOSOME-19
Mammalian genome
QUANTITATIVE TRAIT LOCI MAPPING IN MICE IDENTIFIES X-CHROMOSOME-LINKED CHOLESTEROL GALLSTONE GENES
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CMOAT IS A CANDIDATE GENE FOR LITH2, A CHOLESTEROL GALLSTONE GENE FOUND ON CHROMOSOME-19 IN INBRED MICE
Hepatology
Sterol carrier protein 2 participates in hypersecretion of biliary cholesterol during gallstone formation in genetically gallstone-susceptible mice
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BILIARY SECRETORY IMMUNOGLOBULIN-A IS A MAJOR CONSTITUENT OF THE NEW GROUP OF CHOLESTEROL CRYSTAL-BINDING PROTEINS
Gastroenterology
THE MAJOR GALLSTONE GENE (LITH-1) IS RESPONSIBLE FOR BILIARY CHOLESTEROL (CH) HYPERSECRETION AND CH GALLSTONE FORMATION IN CONGENIC MICE (AKR.L-LITH1(S))
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ESSENTIAL ROLE OF MDR2 P-GLYCOPROTEIN IN REGULATION OF HEPATIC ACTIVITIES OF MAJOR CHOLESTEROL REGULATORY ENZYMES
Gastroenterology
SPONTANEOUS CHOLESTEROL GALLSTONE FORMATION ON NORMAL DIET CHARACTERIZES MICE WITH DISRUPTED MDR2 P-GLYCOPROTEIN GENE AND PROVIDES NEW INSIGHTS INTO CHOLESTEROL CRYSTALLIZATION AND BILE FORMATION
Gastroenterology
EXCESSIVE BILIARY CHOLESTEROL SUPERSATURATION RATHER THAN SOLUBLE PRONUCLEATING PROTEINS OR BILE-SALT HYDROPHOBICITY IS KEY TO HIGH SUSCEPTIBILITY FOR GALLSTONE FORMATION IN THE INBRED MOUSE - A TIME-COURSE STUDY
Gastroenterology
HYPOSECRETION OF BILIARY PHOSPHOLIPIDS (PL) SIGNIFICANTLY DECREASES THE INTESTINAL-ABSORPTION OF CHOLESTEROL (CH) IN MDR2(- -) AND MDR2(+/-) MICE/
Gastroenterology
THERAPEUTIC STRATEGIES IN HILAR BILE-DUCT CARCINOMA (KLATSKIN-TUMOR)
Deutsche Medizinische Wochenschrift
DIAGNOSIS OF HILAR BILE-DUCT CARCINOMA (KLATSKIN TUMOR)
Deutsche Medizinische Wochenschrift
LITH GENES INDUCE OVEREXPRESSION OF STEROL CARRIER PROTEIN 2(SCP2)DURING CHOLESTEROL (CHOL) GALLSTONE FORMATION
The FASEB journal
GENETIC-MAPPING OF HEPATOCANALICULAR TRANSPORTERS ESTABLISHES SISTER-P-GLYCOPROTEIN (SPGP) AS A CANDIDATE FOR THE MAJOR GALLSTONE GENE (LITH1)
Hepatology
QUANTITATIVE TRAIT LOCUS (QTL) MAPPING IDENTIFIES A NEW MURINE GALLSTONE GENE ON CHROMOSOME-17
Hepatology
RAPID CHOLESTEROL (CH) CRYSTALLIZATION IN LITHOGENIC HUMAN BILES IS NOT AFFECTED BY THE PRESENCE OF PURIFIED BILIARY GLYCOPROTEINS
Hepatology
LONG-TERM FEEDING OF CHOLESTEROL (CH) WITHOUT CHOLIC-ACID IS NOT SUFFICIENT TO INDUCE C6 GALLSTONE FORMATION IN INBRED MICE
Hepatology
STEROL CARRIER PROTEIN-2 (SCP2) BUT NOT PHOSPHATIDYLCHOLINE TRANSFER PROTEIN (PCTP) GENE-EXPRESSION IS ENHANCED DURING CHOLESTEROL GALLSTONE FORMATION IN MICE
Gastroenterology
INHIBITION OF CHOLESTEROL CRYSTALLIZATION IS SPECIFIC FOR CRYSTAL-BINDING BILIARY IMMUNOGLOBULIN-A AND NOT A GENERAL CHARACTERISTIC OF HUMAN-IMMUNOGLOBULIN-A
Hepatology
A NEW SUBGROUP OF LECTIN-BOUND BILIARY PROTEINS BINDS TO CHOLESTEROL CRYSTALS, MODIFIES CRYSTAL MORPHOLOGY, AND INHIBITS CHOLESTEROL CRYSTALLIZATION
The Journal of clinical investigation
THE 63 KD AND 28 KD CRYSTAL BINDING-PROTEINS FRACTIONS REPRESENT THE HEAVY AND LIGHT-CHAINS OF HUMAN BILIARY IMMUNOGLOBULIN-A
Gastroenterology
EVIDENCE FOR THE 74 KD INHIBITOR PROTEIN OF CHOLESTEROL CRYSTALLIZATION TO BE THE SECRETORY COMPONENT OF HUMAN BILIARY IMMUNOGLOBULIN-A
Hepatology
ISOLATION OF A NEW POTENT INHIBITOR PROTEIN OF CHOLESTEROL CRYSTAL-GROWTH FROM HUMAN BILE
Hepatology