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Role of cerebral inflammation after traumatic brain injury: A revisited concept
Regulation of chemokines and chemokine receptors after experimental closedhead injury
S-100 beta reflects the extent of injury and outcome, whereas neuronal specific enolase is a better indicator of neuroinflammation in patients with severe traumatic brain injury
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Upregulation of ICAM-1 and MCP-1 but not of MIP-2 and sensorimotor deficitin response to traumatic axonal injury in rats
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