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Involvement of the MLL and RAR alpha genes in a patient with acute monocytic leukemia with t(11;17)(q23;q12)
LEUKEMIA
The t(5;17) acute promyelocytic leukemia fusion protein NPM-RAR interacts with co-repressor and co-activator proteins and exhibits both positive and negative transcriptional properties
BLOOD
Inhibitors of histone deacetylase relieve ETO-mediated repression and induce differentiation of AML1-ETO leukemia cells
CANCER RESEARCH
Chromatin remodeling and leukemia: New therapeutic paradigms
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Overexpression of wild-type retinoic acid receptor alpha (RAR alpha) recapitulates retinoic acid-sensitive transformation of primary myeloid progenitors by acute promyelocytic leukemia RAR alpha-fusion genes
BLOOD
ETO, FUSION PARTNER IN T(8-21) ACUTE MYELOID-LEUKEMIA, REPRESSES TRANSCRIPTION BY INTERACTION WITH THE HUMAN N-COR MSIN3/HDAC1 COMPLEX/
Proceedings of the National Academy of Sciences of the United Statesof America
DIFFERENTIATION OF T(5-17) VARIANT ACUTE PROMYELOCYTIC LEUKEMIC BLASTS BY ALL-TRANS-RETINOIC ACID
Leukemia
ISOLATED DEEP CEREBRAL VENOUS THROMBOSIS TREATED BY DIRECT ENDOVASCULAR THROMBOLYSIS
Surgical neurology
THE T(5-17) APL FUSION PROTEIN NPM-RAR DOES NOT ALTER PML LOCALIZATION
Blood
ATRA INDUCED-DIFFERENTIATION OF T(5-17) APL BLASTS IN-VITRO
Blood
FORCED EXPRESSION OF THE NPM-RAR FUSION GENE INHIBITS DIFFERENTIATIONOF U937 CELLS
Blood
THE T(5-17) VARIANT OF ACUTE PROMYELOCYTIC LEUKEMIA EXPRESSES A NUCLEOPHOSMIN RETINOIC ACID RECEPTOR FUSION
Blood
THE NPM-RAR FUSION PROTEIN ACTS AS A RETINOIC ACID-DEPENDENT TRANSCRIPTIONAL ACTIVATOR
Blood
A NONCLASSICAL TRANSLOCATION INVOLVING 17Q12 (RETINOIC ACID RECEPTOR-ALPHA) IN ACUTE PROMYELOCYTIC LEUKEMIA (APML) WITH ATYPICAL FEATURES
Leukemia
THE T(517) TRANSLOCATION IN ACUTE PROMYELOCYTIC LEUKEMIA GENERATES A NUCLEOPHOSMIN-RARA FUSION TRANSCRIPT
Blood
EXPRESSION OF PML-RAR IN IL-3 DEPENDENT 32D CELLS DOES NOT CONFER FACTOR INDEPENDENCE
Blood