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Titolo:
ADENOSINE-SENSITIVE ATRIAL REENTRANT TACHYCARDIA ORIGINATING FROM THEATRIOVENTRICULAR NODAL TRANSITIONAL AREA
Autore:
IESAKA Y; TAKAHASHI A; GOYA M; SOEJIMA Y; OKAMOTO Y; FUJIWARA H; AONUMA K; NOGAMI A; HIROE M; MARUMO F; HIRAOKA M;
Indirizzi:
TSUCHIURA KYODO HOSP,CTR CARDIOVASC,11-7 MANABESHIN MACHI TSUCHIURA IBARAKI 300 JAPAN TOKYO MED & DENT UNIV,SCH MED,DEPT INTERNAL MED 2 TOKYO 113 JAPAN TOKYO MED & DENT UNIV,MED RES INST,DEPT CARDIOVASC DIS TOKYO 113 JAPAN
Titolo Testata:
Journal of cardiovascular electrophysiology
fascicolo: 8, volume: 8, anno: 1997,
pagine: 854 - 864
SICI:
1045-3873(1997)8:8<854:AARTOF>2.0.ZU;2-P
Fonte:
ISI
Lingua:
ENG
Soggetto:
RADIOFREQUENCY CATHETER ABLATION; PARKINSON-WHITE SYNDROME; SUPRAVENTRICULAR TACHYCARDIA; ELECTROPHYSIOLOGICAL CHARACTERISTICS; ADULT PATIENTS; SLOW-PATHWAY; MECHANISMS; ARRHYTHMIAS;
Keywords:
ATRIAL TACHYCARDIA; ADENOSINE SENSITIVITY; REENTRY; ATRIOVENTRICULAR NODE; AV NODAL TRANSITIONAL TISSUES;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
31
Recensione:
Indirizzi per estratti:
Citazione:
Y. Iesaka et al., "ADENOSINE-SENSITIVE ATRIAL REENTRANT TACHYCARDIA ORIGINATING FROM THEATRIOVENTRICULAR NODAL TRANSITIONAL AREA", Journal of cardiovascular electrophysiology, 8(8), 1997, pp. 854-864

Abstract

Introduction: Atrial tachycardia shows wide variations in its electrophysiologic properties and sites of origin. We report an atrial tachycardia with ECG manifestations and electrophysiologic characteristics similar to an atypical form of AV nodal reentrant tachycardia (AVNRT). Methods and Results: This supraventricular tachycardia was observed in11 patients. It was initiated by atrial extrastimulation with an inverse relationship between the coupling interval of an extrastimulus andthe postextrastimulus interval. Its induction was not related to a jump in the AH interval, and its perpetuation was independent of conduction block in the AV node, Ventricular pacing during tachycardia demonstrated AV dissociation without affecting the atrial cycle length. A very small dose of adenosine triphosphate (mean 3.9 +/- 1.2 mg) could terminate the tachycardia. The earliest atrial activation during tachycardia was recorded at the low anteroseptal right atrium with a different intra-atrial activation sequence from that recorded during ventricular pacing, where the tachycardia was successfully ablated in 9 of 10 attempted patients, Bidirectional AV nodal conduction remained unaffected after successful ablation. Conclusion: There may be an entity of adenosine-sensitive atrial tachycardia probably due to focal reentry within the AV node or its transitional tissues without involvement of theAV nodal pathways. This tachycardia can be ablated without disturbingAV nodal conduction from the right atrial septum.

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Documento generato il 06/07/20 alle ore 04:05:54