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Titolo:
Molecular mechanism of effect of rotating constant magnetic field on organisms
Autore:
Zhang, XY; Luo, ZG; He, XY; Han, QG; Zhang, WD;
Indirizzi:
Shenzhen Univ, Inst Life Sci, Shenzhen 518060, Peoples R China Shenzhen Univ Shenzhen Peoples R China 518060 en 518060, Peoples R China
Titolo Testata:
SCIENCE IN CHINA SERIES C-LIFE SCIENCES
fascicolo: 5, volume: 44, anno: 2001,
pagine: 554 - NIL_1
SICI:
1006-9305(200110)44:5<554:MMOEOR>2.0.ZU;2-Y
Fonte:
ISI
Lingua:
ENG
Soggetto:
RAT ADRENAL-GLAND; NEUROPEPTIDE; STIMULATION; RECEPTOR;
Keywords:
rotating constant magnetic field (RCMF); beta-endorphin; serotonin (5-HT); nitric oxide (NO); neuropeptide Y (NPY);
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Life Sciences
Citazioni:
21
Recensione:
Indirizzi per estratti:
Indirizzo: Zhang, XY Shenzhen Univ, Inst Life Sci, Shenzhen 518060, Peoples R China Shenzhen Univ Shenzhen Peoples R China 518060 Peoples R China
Citazione:
X.Y. Zhang et al., "Molecular mechanism of effect of rotating constant magnetic field on organisms", SCI CHINA C, 44(5), 2001, pp. 554-NIL_1

Abstract

The effect of RCMF-magnetic therapy apparatus on signal substances was studied. The radioimmunoassay (RIA) suggested that the magnetic field increased beta -endorphin markedly in plasma. ELISA indicated that the magnetic field inhibited vomiting reaction induced by chemotherapy drug, with reversible decrease of serotonin (5-HT) level in brains, small intestine tissue and serum. Furthermore, the bioeffect of magnetic fields on 5-HT level presented a typical window effect and post-effect, and the inhibitory effect of magnetic field on the emesis was parallel to the decrease level of 5-HT. This result implied that the decrease of 5-HT might be the basis of rotating constant magnetic field (RCMF) inhibiting drug-induced emesis. The nitric acidreductase-spectrophotometry and nicotinamide adenine dinucleotide-diaphorase/arginine-vasopressin (AVP) cytochemistry technique showed that the magnetic field induced nitric oxide (NO) increase in hypothalamus and the high NO(A) level lasted for 3 hours. The results suggested that NO(A) increases after the treatment of the magnetic field in hypothalamus, which may result from strong expression of NO-ergic neuron in paraventricular hypothalamic nucleus (PVN), periventricular hypothalamic nucleus (PEN) and supraoptic nucleus (SON). The coexistence of NO and AVP may play an important role in theregulation of endocrine and neuroendocrine by the magnetic field. And our data also confirmed that the magnetic field increased the content of NO so strongly that high NO level lasted for 3 hours, also made neuropeptide Y (NPY) cell in medulla stained heavily.

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Documento generato il 08/04/20 alle ore 23:54:38