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Titolo:
Clinically silent circulating microemboli in 20 patients with carotid or vertebral artery dissection
Autore:
Droste, DW; Junker, K; Stogbauer, F; Lowens, S; Besselmann, M; Braun, B; Ringelstein, EB;
Indirizzi:
Univ Munster, Dept Neurol, D-4400 Munster, Germany Univ Munster Munster Germany D-4400 Dept Neurol, D-4400 Munster, Germany Univ Munster, Dept Radiol, D-4400 Munster, Germany Univ Munster Munster Germany D-4400 Dept Radiol, D-4400 Munster, Germany
Titolo Testata:
CEREBROVASCULAR DISEASES
fascicolo: 3, volume: 12, anno: 2001,
pagine: 181 - 185
SICI:
1015-9770(2001)12:3<181:CSCMI2>2.0.ZU;2-F
Fonte:
ISI
Lingua:
ENG
Soggetto:
TRANSCRANIAL DOPPLER; EMBOLI;
Keywords:
ultrasonography; cerebral blood flow; embolism; dissection;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Life Sciences
Citazioni:
14
Recensione:
Indirizzi per estratti:
Indirizzo: Droste, DW WWU Munster, Neurol Klin & Poliklin, Alberth Schweitzer Str 33,D-18129 Munster, Germany WWU Munster Alberth Schweitzer Str 33 Munster Germany D-18129
Citazione:
D.W. Droste et al., "Clinically silent circulating microemboli in 20 patients with carotid or vertebral artery dissection", CEREB DIS, 12(3), 2001, pp. 181-185

Abstract

Background and Purpose: Carotid and vertebral artery dissections are frequently complicated by cerebral embolism. Detection of clinically silent circulating microemboli by transcranial Doppler sonography (TCD) is now widely investigated in patients with carotid artery disease in the hope to identify patients at increased risk for stroke. Methods: In 20 patients with carotid (n = 17) or vertebral (n = 2) artery dissection, or both (n = 1), we performed a 1-hour microembolus detection downstream to the dissection in the middle or in the posterior cerebral artery, respectively. Results: Five patients with a carotid artery stenosis of greater than or equal to 90% or with carotid artery occlusion showed microembolic signals at a rate Of LIP to 15 events/h. In all these patients, the onset of the dissection was within the last 58 days. Patients with lower degrees of stenosis or onset of symptoms beyond 58 days did not show microembolic signals at all. Three patientswho had presented with recurrent ischaemic events prior to TCD monitoring unexceptionally had microembolic signals. Conclusion: Microembolic signals occur in patients with high-grade stenosis or occlusion due to acute cervical artery dissection. Patients with microemboli seem to be at increased macroembolic risk, i.e. stroke recurrence, and may require close-meshed clinical follow-up and possibly stronger antithrombotic treatment. Copyright (C) 2001 S.Karger AG, Basel.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 23/01/21 alle ore 22:29:30