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Titolo:
An immediate-shock freezing deficit with discrete cues: A possible role for unconditioned stimulus processing mechanisms
Autore:
Lattal, KM; Abel, T;
Indirizzi:
Univ Penn, Dept Biol, Philadelphia, PA 19104 USA Univ Penn Philadelphia PA USA 19104 Dept Biol, Philadelphia, PA 19104 USA
Titolo Testata:
JOURNAL OF EXPERIMENTAL PSYCHOLOGY-ANIMAL BEHAVIOR PROCESSES
fascicolo: 4, volume: 27, anno: 2001,
pagine: 394 - 406
SICI:
0097-7403(200110)27:4<394:AIFDWD>2.0.ZU;2-K
Fonte:
ISI
Lingua:
ENG
Soggetto:
DEFENSIVE REACTIONS; LATENT INHIBITION; RATS; HIPPOCAMPUS; ENVIRONMENT; PREEXPOSURE; ANALGESIA; INTERVAL; BEHAVIOR; EXPOSURE;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Social & Behavioral Sciences
Life Sciences
Citazioni:
36
Recensione:
Indirizzi per estratti:
Indirizzo: Abel, T Univ Penn, Dept Biol, 3740 Hamilton Walk, Philadelphia, PA 19104 USA Univ Penn 3740 Hamilton Walk Philadelphia PA USA 19104 A 19104 USA
Citazione:
K.M. Lattal e T. Abel, "An immediate-shock freezing deficit with discrete cues: A possible role for unconditioned stimulus processing mechanisms", J EXP PSY A, 27(4), 2001, pp. 394-406

Abstract

Five experiments with C57BL/6 mice (Mus musculus) investigated whether failures in shock processing might contribute to deficits in freezing that occur after an animal receives a shock immediately on exposure to a conditioning context. Experiment 1 found that more contextual freezing resulted from delayed shocks than from immediate shocks across 4 shock intensities. Experiment 2 extended the immediate-shock freezing deficit to discrete stimuli. Experiment 3 found that preexposure to the to-be-conditioned cue did not facilitate immediate cued conditioning. Experiment 4 found that context preexposure enhanced context-evoked fear after an immediate shock. Experiment 5 found that context preexposure also enhanced immediate cued conditioning. These findings are problematic for current theories of the immediate-shock freezing deficit that focus exclusively on processing of the conditioned stimulus, and they suggest that failures in shock processing may contribute tothe deficit.

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Documento generato il 21/01/20 alle ore 06:53:51