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Titolo:
Transient crossed cerebellar diaschisis following thalamic hemorrhage
Autore:
Takasawa, M; Hashikawa, K; Ohtsuki, T; Imaizumi, M; Oku, N; Kitagawa, K;
Indirizzi:
Osaka Univ, Grad Sch Med, Dept Internal Med & Therapeut, Suita, Osaka 5650871, Japan Osaka Univ Suita Osaka Japan 5650871 erapeut, Suita, Osaka 5650871, Japan Osaka Univ, Grad Sch Med, Dept Nucl Med, Suita, Osaka 5650871, Japan OsakaUniv Suita Osaka Japan 5650871 ucl Med, Suita, Osaka 5650871, Japan
Titolo Testata:
JOURNAL OF NEUROIMAGING
fascicolo: 4, volume: 11, anno: 2001,
pagine: 438 - 440
SICI:
1051-2284(200110)11:4<438:TCCDFT>2.0.ZU;2-Q
Fonte:
ISI
Lingua:
ENG
Soggetto:
SPECT;
Keywords:
brain hemorrhage; crossed cerebellar diaschisis; Tc-99m-hexamethylpropyleneamine oxime; single photon emission computed tomography;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Citazioni:
10
Recensione:
Indirizzi per estratti:
Indirizzo: Takasawa, M Osaka Univ, Grad Sch Med, Dept Internal Med & Therapeut, 2-2 Yamadaoka, Suita, Osaka 5650871, Japan Osaka Univ 2-2 Yamadaoka Suita Osaka Japan 5650871 0871, Japan
Citazione:
M. Takasawa et al., "Transient crossed cerebellar diaschisis following thalamic hemorrhage", J NEUROIMAG, 11(4), 2001, pp. 438-440

Abstract

This report concerns a 65-year-old right-handed woman with cerebral hemorrhage who presented with mild right-sided hemiparesis. Computed tomography (CT) revealed hematoma in the left thalamus and compression of the posteriorlimb of the internal capsule by a brain edema surrounding the lesion. Tc-99m-hexamethylpropyleneamine oxime (HMPAO) single photon emission computed tomography (SPECT) images obtained 4 days after onset showed hypoperfusion in the left thalamus containing a hematoma as well as contralateral cerebellar hypoperfusion to the supratentorial lesion, which is well recognized as crossed cerebellar diaschisis (CCD) after stroke. CT 14 days after the onset revealed reduction of the brain edema of the posterior limb of the internal capsule accompanied by gradual neurological improvement. SPECT obtained 14 and 28 days later showed that CCD had disappeared. In this case report, the authors discuss the disappearance of CCD due to transient edematous compression of the internal capsule following thalamic hemorrhage on serial Tc-99m-HMPAO SPECT scans. CCD was possibly caused by the lesion confined to the posterior limb of the internal capsule, which anatomically constitutes the cerebro-pontocerebellar pathway.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 17/09/19 alle ore 22:39:35