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Titolo:
Regulation of Rho GTPases by p120-catenin
Autore:
Anastasiadis, PZ; Reynolds, AB;
Indirizzi:
Vanderbilt Univ, Dept Canc Biol, Nashville, TN 37232 USA Vanderbilt Univ Nashville TN USA 37232 Canc Biol, Nashville, TN 37232 USA
Titolo Testata:
CURRENT OPINION IN CELL BIOLOGY
fascicolo: 5, volume: 13, anno: 2001,
pagine: 604 - 610
SICI:
0955-0674(200110)13:5<604:RORGBP>2.0.ZU;2-X
Fonte:
ISI
Lingua:
ENG
Soggetto:
CELL-CELL ADHESION; E-CADHERIN; P120 CATENIN; ACTIN CYTOSKELETON; FAMILY GTPASES; ALPHA-CATENIN; N-CADHERIN; PROTEIN; P120(CTN); RAC;
Tipo documento:
Review
Natura:
Periodico
Settore Disciplinare:
Life Sciences
Citazioni:
46
Recensione:
Indirizzi per estratti:
Indirizzo: Anastasiadis, PZ Vanderbilt Univ, Dept Canc Biol, 1161 21st Ave S,MCN C-2310, Nashville, TN37232 USA Vanderbilt Univ 1161 21st Ave S,MCN C-2310 Nashville TN USA 37232
Citazione:
P.Z. Anastasiadis e A.B. Reynolds, "Regulation of Rho GTPases by p120-catenin", CURR OP CEL, 13(5), 2001, pp. 604-610

Abstract

Three recent reports indicate that p120-catenin can modulate the activities of RhoA, Rac and Cdc42, suggesting an elegant and previously unexpected mechanism for regulating the balance between adhesive and motile cellular phenotypes. The observations in these reports provide important new clues toward p120's mechanism of action and provide a potential explanation for the metastatic phenotype exhibited in carcinoma cells that have lost E cadherinexpression.

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Documento generato il 04/12/20 alle ore 13:06:25