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Titolo:
NEURALLY-MEDIATED SYNCOPE - A REVIEW OF CARDIAC AND ARTERIAL RECEPTORS
Autore:
GRIMM DR;
Indirizzi:
VET AFFAIRS MED CTR,SPINAL CORD DAMAGE RES CTR,RM 1E-02,130 W KINGSBRIDGE RD BRONX NY 10468 MT SINAI MED CTR,DEPT MED NEW YORK NY 10029
Titolo Testata:
Journal of clinical neurophysiology
fascicolo: 3, volume: 14, anno: 1997,
pagine: 170 - 182
SICI:
0736-0258(1997)14:3<170:NS-ARO>2.0.ZU;2-6
Fonte:
ISI
Lingua:
ENG
Soggetto:
LEFT-VENTRICULAR RECEPTORS; HEAD-UP TILT; VAGAL C-FIBERS; RAT AORTIC BARORECEPTORS; SYMPATHETIC-NERVE FIBERS; LEFT ATRIAL DISTENSION; PLASMA-RENIN ACTIVITY; AFFERENT-FIBERS; BLOOD-PRESSURE; ANESTHETIZED DOGS;
Keywords:
ATRIAL AND VENTRICULAR RECEPTORS; SYMPATHETIC AND PARASYMPATHETIC NERVOUS SYSTEM; ARTERIAL BARORECEPTORS; NEUROCARDIOGENIC SYNCOPE;
Tipo documento:
Review
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Science Citation Index Expanded
Citazioni:
141
Recensione:
Indirizzi per estratti:
Citazione:
D.R. Grimm, "NEURALLY-MEDIATED SYNCOPE - A REVIEW OF CARDIAC AND ARTERIAL RECEPTORS", Journal of clinical neurophysiology, 14(3), 1997, pp. 170-182

Abstract

This article reviews the basic physiology of the reflexogenic areas of the heart and the mechano- and baroreceptors that regulate cardiovascular and autonomic homeostasis, all of which contribute to our understanding of the pathogenesis of neurally mediated syncope. The mechanisms of neurally mediated syncope may involve excessive activation of ventricular receptors that trigger severe hypotension and bradycardia. Thus, neurally mediated syncope may be the clinical expression of the Bezold-Jarisch reflex, which occurs in situations of increased sympathetic activity, perhaps as a result of heightened cardiac receptor sensitivity. The arterial baroreceptors exert a ubiquitous influence on theheart and circulation, and serve primarily to buffer transient changes in arterial pressure by transmitting sensory information regarding their stretch to the central nervous system. This information, in conjunction with cardiac receptor input, elicits alterations in neural efferent output from sympathetic and parasympathetic fibers to provide subtle, continuous regulation of beat-to-beat cardiovascular hemodynamicsto an array of physiologic and psychological stressors.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 24/11/20 alle ore 22:07:14