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Titolo:
Host-pathogen interactions promoting inflammatory Lyme arthritis: use of mouse models for dissection of disease processes
Autore:
Wooten, RM; Weis, JJ;
Indirizzi:
Univ Utah, Sch Med, Dept Pathol, Div Cell Biol & Immunol, Salt Lake City, UT 84132 USA Univ Utah Salt Lake City UT USA 84132 munol, Salt Lake City, UT 84132 USA
Titolo Testata:
CURRENT OPINION IN MICROBIOLOGY
fascicolo: 3, volume: 4, anno: 2001,
pagine: 274 - 279
SICI:
1369-5274(200106)4:3<274:HIPILA>2.0.ZU;2-F
Fonte:
ISI
Lingua:
ENG
Soggetto:
BORRELIA-BURGDORFERI LIPOPROTEINS; TOLL-LIKE RECEPTOR-2; OUTER SURFACE PROTEIN; TREPONEMA-PALLIDUM; PATHWAY DISTINCT; MONOCYTIC CELLS; KAPPA-B; MICE; LIPOPOLYSACCHARIDE; ACTIVATION;
Tipo documento:
Review
Natura:
Periodico
Settore Disciplinare:
Agriculture,Biology & Environmental Sciences
Life Sciences
Citazioni:
71
Recensione:
Indirizzi per estratti:
Indirizzo: Wooten, RM Univ Utah, Sch Med, Dept Pathol, Div Cell Biol & Immunol, 50 N Med Dr, Salt Lake City, UT 84132 USA Univ Utah 50 N Med Dr Salt Lake City UT USA 84132 UT 84132 USA
Citazione:
R.M. Wooten e J.J. Weis, "Host-pathogen interactions promoting inflammatory Lyme arthritis: use of mouse models for dissection of disease processes", CURR OPIN M, 4(3), 2001, pp. 274-279

Abstract

Recent studies have confirmed the infectious and inflammatory nature of arthritis induced by Borrelia burgdorferi, or Lyme arthritis. This arthritis is directed by the presence of the bacteria in joint tissue, and is mediated through activation of the Toll-like receptor 2 (TLR2) signaling pathways by borrelial lipoproteins. Several host genes regulate the severity of arthritis. possibly by regulating the balance of pro- and antiinflammatory responses.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 01/04/20 alle ore 16:36:29