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Titolo:
BLOOD-PRESSURE, GENDER, AND PARENTAL HYPERTENSION ARE FACTORS IN BASE-LINE AND POSTSTRESS PAIN SENSITIVITY IN NORMOTENSIVE ADULTS
Autore:
BRAGDON EE; LIGHT KC; GIRDLER SS; MAIXNER W;
Indirizzi:
UNIV N CAROLINA,DEPT PSYCHOL,STRESS & HLTH RES PROGRAM,CB 7175,BLDG A,MED RES BLDG CHAPEL HILL NC 27599 UNIV N CAROLINA,DEPT PSYCHOL,STRESS & HLTH RES PROGRAM CHAPEL HILL NC27599 UNIV N CAROLINA,DEPT PSYCHIAT CHAPEL HILL NC 27599 UNIV N CAROLINA,DENT RES CTR CHAPEL HILL NC 27599
Titolo Testata:
International journal of behavioral medicine
fascicolo: 1, volume: 4, anno: 1997,
pagine: 17 - 38
SICI:
1070-5503(1997)4:1<17:BGAPHA>2.0.ZU;2-F
Fonte:
ISI
Lingua:
ENG
Soggetto:
SEX-DIFFERENCES; CARDIOVASCULAR REACTIVITY; BARORECEPTOR STIMULATION; THERMAL SENSITIVITY; INDUCED ANALGESIA; SWIM ANALGESIA; DENTAL PAIN; PERCEPTION; RATS; HUMANS;
Keywords:
ANALGESIA; BLOOD PRESSURE; CARDIAC INDEX; GENDER; PARENTAL HYPERTENSION; STRESS;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Physical, Chemical & Earth Sciences
Citazioni:
65
Recensione:
Indirizzi per estratti:
Citazione:
E.E. Bragdon et al., "BLOOD-PRESSURE, GENDER, AND PARENTAL HYPERTENSION ARE FACTORS IN BASE-LINE AND POSTSTRESS PAIN SENSITIVITY IN NORMOTENSIVE ADULTS", International journal of behavioral medicine, 4(1), 1997, pp. 17-38

Abstract

We studied 38 men and 36 women to learn whether a brief speech stressor reduced normotensive humans' thermal pain sensitivity, whether baseline and poststress pain threshold and tolerance varied with blood pressure (BP) and hemodynamic measures, and whether these relations differed by gender and parental hypertension (PH). PH-women with low resting BPs had lower baseline pain tolerance than did ail other groups tps <.05), and this group alone exhibited stress-induced analgesia (p =.008). In women, pre- and poststress pain tolerance varied directly with rest and stress BP tps <.05). In men, BP and pain measures were not related, but high cardiac index during stress was associated with low baseline pain tolerance (p <.01). The present findings support the hypothesis that pain sensitivity and cardiac stress response share a commonmechanism, but they yield little support for the hypothesis that analgesic responses to acute stress contribute to hypertension etiology via an instrumental learning process.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 29/09/20 alle ore 04:07:45