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Titolo:
Recombination between paralogues at the rp1 rust resistance locus in maize
Autore:
Sun, Q; Collins, NC; Ayliffe, M; Smith, SM; Drake, J; Pryor, T; Hulbert, SH;
Indirizzi:
Kansas State Univ, Dept Plant Pathol, Manhattan, KS 66506 USA Kansas StateUniv Manhattan KS USA 66506 Pathol, Manhattan, KS 66506 USA John Innes Ctr, Sainsbury Lab, Norwich NR4 7UH, Norfolk, England John Innes Ctr Norwich Norfolk England NR4 7UH NR4 7UH, Norfolk, England CSIRO, Div Plant Ind, Canberra, ACT 2601, Australia CSIRO Canberra ACT Australia 2601 lant Ind, Canberra, ACT 2601, Australia
Titolo Testata:
GENETICS
fascicolo: 1, volume: 158, anno: 2001,
pagine: 423 - 438
SICI:
0016-6731(200105)158:1<423:RBPATR>2.0.ZU;2-9
Fonte:
ISI
Lingua:
ENG
Soggetto:
LEUCINE-RICH REPEAT; PLANT-DISEASE RESISTANCE; DOWNY MILDEW RESISTANCE; GENE-CLUSTER; DIVERSIFYING SELECTION; PATHOGEN RESISTANCE; MEIOTIC INSTABILITY; ADAPTIVE EVOLUTION; 6 HOMOLOGS; TOMATO;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Agriculture,Biology & Environmental Sciences
Life Sciences
Citazioni:
58
Recensione:
Indirizzi per estratti:
Indirizzo: Hulbert, SH Kansas State Univ, Dept Plant Pathol, Throckmorton Hall, Manhattan, KS 66506 USA Kansas State Univ Throckmorton Hall Manhattan KS USA 66506 USA
Citazione:
Q. Sun et al., "Recombination between paralogues at the rp1 rust resistance locus in maize", GENETICS, 158(1), 2001, pp. 423-438

Abstract

Rp1 is a complex rust resistance locus of maize. The HRp1-D haplotype is composed of Rp1-D and eight paralogues, seven of which also code fur predicted nucleotide binding site-leucine rich repeat (NBS-LRR) proteins similar to the Rp1-D gene. The paralogues are polymorphic (DNA identities 91-97%), especially in the C-terminal LRR domain. The remaining family member encodesa truncated protein that has no LRR domain. Seven of the nine family members, including the truncated gene, are transcribed. Sequence comparisons between paralogues provide evidence for past recombination events between paralogues and diversifying selection, particularly in the C-terminal half of tile LRR domain. Variants selected for complete or partial loss of Rp1-D resistance can he explained by unequal crossing over that occurred mostly within coding regions. The Rp1-D gene is altered or lost in all variants, the recombination breakpoints occur throughout the genes, and most recombinant events (9/14 examined) involved the same untranscribed paralogue with the Rp1-D gene. One recombinant with a complete LRR from Rp1-D, hut the aminoterminal portion from another homologue, conferred the Rp1-D specificity but with a reduced level of resistance.

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Documento generato il 19/09/20 alle ore 08:48:30