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Titolo:
The changing pattern of diagnosis of infantile cholestasis
Autore:
Stormon, MO; Dorney, SFA; Kamath, KR; OLoughlin, EV; Gaskin, KJ;
Indirizzi:
Royal Alexandra Hosp Children, Dept Gastroenterol, Parramatta, NSW 2124, Australia Royal Alexandra Hosp Children Parramatta NSW Australia 2124 24, Australia
Titolo Testata:
JOURNAL OF PAEDIATRICS AND CHILD HEALTH
fascicolo: 1, volume: 37, anno: 2001,
pagine: 47 - 50
SICI:
1034-4810(200102)37:1<47:TCPODO>2.0.ZU;2-C
Fonte:
ISI
Lingua:
ENG
Soggetto:
LIVER-DISEASE; CONGENITAL HYPOPITUITARISM; NEONATAL CHOLESTASIS; BILIARY ATRESIA; DEFICIENCY; HEPATITIS; CHILDREN;
Keywords:
biliary obstruction; bile duct hypoplasia; cholestasis; genetic metabolic disease; liver disease; neonatal;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Citazioni:
18
Recensione:
Indirizzi per estratti:
Indirizzo: Gaskin, KJ Royal Alexandra Hosp Children, Dept Gastroenterol, POB 3515, Parramatta, NSW 2124, Australia Royal Alexandra Hosp Children POB 3515 Parramatta NSW Australia 2124
Citazione:
M.O. Stormon et al., "The changing pattern of diagnosis of infantile cholestasis", J PAEDIAT C, 37(1), 2001, pp. 47-50

Abstract

Objective: Cholestatic liver disease in infancy is caused by a wide range of conditions. This study reviews the pattern of diagnosis of infants with cholestasis presenting to a tertiary referral paediatric hospital in Sydney, Australia, during a 12-year period (1985-96). Methodology: Infants aged less than 6 months with cholestasis were identified retrospectively from hospital records and data retrieved from the medical records. Results: There were 205 infants identified as having cholestatic liver disease. The aetiology of the cholestasis was idiopathic in 25%, metabolic/genetic in 23%, and due to obstruction in 20%, parenteral nutrition in 20%, infection in 9% and bile duct hypoplasia in 3%. Conclusions: This study highlights the changing patterns of diagnosis of cholestatic liver disease in infants at a tertiary paediatric facility, demonstrating that up to 50% of cases are now due to genetic/metabolic diseasesor parenteral nutrition, and a high proportion are due to idiopathic disease.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 19/09/20 alle ore 18:42:57