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Titolo:
Basic mechanisms of reentrant arrhythmias
Autore:
Antzelevitch, C;
Indirizzi:
Mason Med Res Lab, Utica, NY 13501 USA Mason Med Res Lab Utica NY USA 13501 son Med Res Lab, Utica, NY 13501 USA
Titolo Testata:
CURRENT OPINION IN CARDIOLOGY
fascicolo: 1, volume: 16, anno: 2001,
pagine: 1 - 7
SICI:
0268-4705(200101)16:1<1:BMORA>2.0.ZU;2-Z
Fonte:
ISI
Lingua:
ENG
Soggetto:
LONG-QT SYNDROME; TORSADE-DE-POINTES; CANINE VENTRICULAR EPICARDIUM; RABBIT ATRIAL MUSCLE; ST-SEGMENT ELEVATION; M-CELLS; CELLULAR BASIS; BRUGADA-SYNDROME; CIRCUS MOVEMENT; ELECTROPHYSIOLOGIC CHARACTERISTICS;
Tipo documento:
Review
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Citazioni:
78
Recensione:
Indirizzi per estratti:
Indirizzo: Antzelevitch, C Mason Med Res Lab, 2150 Bleecker St, Utica, NY 13501 USA Mason Med Res Lab 2150 Bleecker St Utica NY USA 13501 USA
Citazione:
C. Antzelevitch, "Basic mechanisms of reentrant arrhythmias", CURR OPIN C, 16(1), 2001, pp. 1-7

Abstract

The mechanisms responsible for active cardiac arrhythmias are generally divided into two major categories: (1) enhanced or abnormal impulse formationand (2) reentry. Reentry can be subdivided into three subcategories: (1) circus movement, (2) reflection, and (3) Phase 2 reentry. Reentry occurs when a propagating impulse fails to die out after normal activation of the heart and persists to re-excite the heart after expiration of the refractory period. Evidence implicating reentry as a mechanism of cardiac arrhythmias stems back to the turn of the century. Amplification of intrinsic electricalheterogeneities provides the substrate responsible for developing Phase 2 and circus movement reentry, which underlie Ventricular tachycardia in the long QT and Brugada syndromes. Curr Opin Cardiol 2001, 16:1-7 (C) 2001 Lippincott Williams & Wilkins, Inc.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 31/03/20 alle ore 18:57:12