Catalogo Articoli (Spogli Riviste)

OPAC HELP

Titolo:
Transient expression of synaptogyrin in the ganglionic eminence of the human fetal brain
Autore:
Ulfig, N; Feldhaus, C; Bohl, J;
Indirizzi:
Univ Rostock, Dept Anat, Neuroembryon Res Lab, D-18055 Rostock, Germany Univ Rostock Rostock Germany D-18055 n Res Lab, D-18055 Rostock, Germany Univ Mainz, Dept Neuropathol, D-55131 Mainz, Germany Univ Mainz Mainz Germany D-55131 ept Neuropathol, D-55131 Mainz, Germany
Titolo Testata:
ANNALS OF ANATOMY-ANATOMISCHER ANZEIGER
fascicolo: 6, volume: 182, anno: 2000,
pagine: 505 - 508
SICI:
0940-9602(200011)182:6<505:TEOSIT>2.0.ZU;2-9
Fonte:
ISI
Lingua:
ENG
Soggetto:
INTERMEDIATE TARGET; SYNAPTOPHYSIN; PROTEIN; NEURONS; CELLS; AXONS; P-29;
Keywords:
pioneer axons; development; internal capsule; immunohistochemistry; synaptogyrin; axonal guidance;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Life Sciences
Citazioni:
15
Recensione:
Indirizzi per estratti:
Indirizzo: Ulfig, N Univ Rostock, Dept Anat, Neuroembryon Res Lab, Gertrudenstr 9, D-18055 Rostock, Germany Univ Rostock Gertrudenstr 9 Rostock Germany D-18055ock, Germany
Citazione:
N. Ulfig et al., "Transient expression of synaptogyrin in the ganglionic eminence of the human fetal brain", ANN ANATOMY, 182(6), 2000, pp. 505-508

Abstract

The ganglionic eminence (GE) representing a conspicuous bulb-like elevation of the telencephalic proliferative zone has recently been shown to be involved in the establishment of cortical connections. This study demonstrate s the presence of synaptogyrin-immunoreactivity in a large number of cell bodies of the human GE between 12 and 20 weeks of gestation. From the 20(th)week onwards synaptogyrin expression sharply declines. No immunoreactive structures are detectable in the 23(rd) week or later. As the CE persists nearly throughout the entire fetal period these results show that its neurochemical features change distinctly in the course of development. The synaptogyrin-immunoreactive GE-cells may form an early corticopedal connection which provides a scaffold for outgrowing cortical axons.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 04/04/20 alle ore 21:46:28