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Titolo:
The Arabidopsis sugar-insensitive mutants sis4 and sis5 are defective in abscisic acid synthesis and response
Autore:
Laby, RJ; Kincaid, MS; Kim, DG; Gibson, SI;
Indirizzi:
Rice Univ, Dept Biochem & Cell Biol, Houston, TX 77005 USA Rice Univ Houston TX USA 77005 Biochem & Cell Biol, Houston, TX 77005 USA
Titolo Testata:
PLANT JOURNAL
fascicolo: 5, volume: 23, anno: 2000,
pagine: 587 - 596
SICI:
0960-7412(200009)23:5<587:TASMSA>2.0.ZU;2-8
Fonte:
ISI
Lingua:
ENG
Soggetto:
REGULATED GENE-EXPRESSION; HIGHER-PLANTS; ASPARAGINE SYNTHETASE; METABOLIC-REGULATION; SIGNAL-TRANSDUCTION; SUCROSE SYNTHASE; PROTEIN-KINASES; BETA-AMYLASE; WD PROTEIN; THALIANA;
Keywords:
Arabidopsis; sis; abscisic acid; mutant; insensitive; sugar;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Agriculture,Biology & Environmental Sciences
Life Sciences
Citazioni:
64
Recensione:
Indirizzi per estratti:
Indirizzo: Gibson, SI Rice Univ, Dept Biochem & Cell Biol, MS140,6100 Main St, Houston, TX 77005USA Rice Univ MS140,6100 Main St Houston TX USA 77005 , TX 77005USA
Citazione:
R.J. Laby et al., "The Arabidopsis sugar-insensitive mutants sis4 and sis5 are defective in abscisic acid synthesis and response", PLANT J, 23(5), 2000, pp. 587-596

Abstract

Although soluble sugar levels affect many aspects of plant development andphysiology, little is known about the mechanisms by which plants respond to sugar. Here we report the isolation of 13 sugar-insensitive (sis) mutantsof Arabidopsis that, unlike wild-type plants, are able to form expanded cotyledons and true leaves when germinated on media containing high concentrations of glucose or sucrose. The sis4 and sis5 mutants are allelic to the ABA-biosynthesis mutant aba2 and the ABA-insensitive mutant abi4, respectively. In addition to being insensitive to glucose and sucrose, the sis4/aba2 and sis5/abi4 mutants also display decreased sensitivity to the inhibitory effects of mannose on early seedling development. Mutations in the ABI5 gene, but not mutations in the ABI1, ABI2 or ABI3 genes, also lead to weak glucose- and mannose-insensitive phenotypes. Wild-type and mutant plants show similar responses to the effects of exogenous sugar on chlorophyll and anthocyanin accumulation, indicating that the mutants are not defective in all sugar responses. These results indicate that defects in ABA metabolism and some, but not all, defects in ABA response can also alter response to exogenous sugar.

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Documento generato il 25/09/20 alle ore 13:42:41