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Titolo:
Active ion secretion and permeability of rabbit maxillary sinus epithelium, impact of staphylococcal enterotoxin B
Autore:
Yang, PC; Liu, T; Wang, CS; Zhang, NZ; Tao, ZD;
Indirizzi:
Shanxi Med Unit, Affiliated Hosp 1, Dept Otolaryngol, Taiyuan, Peoples R China Shanxi Med Unit Taiyuan Peoples R China yngol, Taiyuan, Peoples R China Hunan Med Univ, Yale Xiang Hosp, Changsha, Peoples R China Hunan Med UnivChangsha Peoples R China Hosp, Changsha, Peoples R China
Titolo Testata:
OTOLARYNGOLOGY-HEAD AND NECK SURGERY
fascicolo: 1, volume: 123, anno: 2000,
parte:, 1
pagine: 120 - 123
SICI:
0194-5998(200007)123:1<120:AISAPO>2.0.ZU;2-G
Fonte:
ISI
Lingua:
ENG
Soggetto:
TUMOR-NECROSIS-FACTOR; SUPERANTIGENS; TRANSPORT;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Citazioni:
16
Recensione:
Indirizzi per estratti:
Indirizzo: Yang, PC McMaster Univ, Hlth Sci Ctr, Room 3N5C,1200 Main St W, Hamilton, ON L8N 3Z5, Canada McMaster Univ Room 3N5C,1200 Main St W Hamilton ON Canada L8N 3Z5
Citazione:
P.C. Yang et al., "Active ion secretion and permeability of rabbit maxillary sinus epithelium, impact of staphylococcal enterotoxin B", OTO H N SUR, 123(1), 2000, pp. 120-123

Abstract

The aim of this study was to investigate the impact of staphylococcal enterotoxin B (SEB), a model superantigen, on the physiologic functions of rabbit maxillary sinus epithelium. Rabbit sinus mucosae were separated under a surgical microscope and mounted in Ussing chambers to record short-circuit current, conductance, and permeability to horseradish peroxidase. The results showed that SEE evoked increases in sinus epithelial cell baseline short-circuit current, conductance, and permeability to horseradish peroxidase, When tumor necrosis factor-alpha (TNF-alpha) was added to the Ussing chambers, we got results similar to those obtained by SEE stimulation in vitro; the effects of SEE on sinus epithelial cells could be blocked by pretreatment with anti-TNF-alpha antibody. These results demonstrate that SEE is able to alter the function of sinus epithelial cells and to affect the capability of the epithelial defensive barrier, which may be mediated by TNF-alpha.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 14/11/18 alle ore 04:19:32