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Titolo:
History of zinc as related to brain function
Autore:
Sandstead, HH; Frederickson, CJ; Penland, JG;
Indirizzi:
Univ Texas, Med Branch, Dept Prevent Med & Community Hlth, Galveston, TX 77555 USA Univ Texas Galveston TX USA 77555 Community Hlth, Galveston, TX 77555 USA Univ Texas, Med Branch, Ctr Bioengn, Galveston, TX 77555 USA Univ Texas Galveston TX USA 77555 h, Ctr Bioengn, Galveston, TX 77555 USA Univ Texas, Med Branch, Dept Neurosci, Galveston, TX 77555 USA Univ TexasGalveston TX USA 77555 Dept Neurosci, Galveston, TX 77555 USA NeuroBioTex Inc, Galveston, TX 77550 USA NeuroBioTex Inc Galveston TX USA77550 ioTex Inc, Galveston, TX 77550 USA ARS, USDA, Grand Forks Human Nutr Res Ctr, Grand Forks, ND 58201 USA ARS Grand Forks ND USA 58201 uman Nutr Res Ctr, Grand Forks, ND 58201 USA
Titolo Testata:
JOURNAL OF NUTRITION
fascicolo: 2, volume: 130, anno: 2000, supplemento:, S
pagine: 496S - 502S
SICI:
0022-3166(200002)130:2<496S:HOZART>2.0.ZU;2-Y
Fonte:
ISI
Lingua:
ENG
Soggetto:
HIPPOCAMPAL MOSSY FIBERS; DEVELOPING RAT-BRAIN; METHYL-D-ASPARTATE; CORTICAL-NEURONS; DIETARY ZINC; KAINIC ACID; FOOD-INTAKE; COGNITIVE PERFORMANCE; METALLOTHIONEIN-III; CONFERS RESISTANCE;
Keywords:
zinc; brain; hippocampus; neurotransmission; cognition;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Agriculture,Biology & Environmental Sciences
Life Sciences
Citazioni:
161
Recensione:
Indirizzi per estratti:
Indirizzo: Sandstead, HH Univ Texas, Med Branch, Dept Prevent Med & Community Hlth, Galveston, TX 77555 USA Univ Texas Galveston TX USA 77555 , Galveston, TX 77555 USA
Citazione:
H.H. Sandstead et al., "History of zinc as related to brain function", J NUTR, 130(2), 2000, pp. 496S-502S

Abstract

Zinc (Zn) is essential for synthesis of coenzymes that mediate biogenic-amine synthesis and metabolism. Zn from vesicles in presynaptic terminals of certain glutaminergic neurons modulates postsynaptic N-methyl-D-aspartate (NMDA) receptors for glutamate. Large amounts of Zn released from vesicles by seizures or ischemia can kill postsynaptic neurons. Acute Zn deficiency impairs brain function of experimental animals and humans. Zn deficiency in experimental animals during early brain development causes malformations, whereas deficiency later in brain development causes microscopic abnormalities and impairs subsequent function. A limited number of studies suggest that similar phenomena can occur in humans.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 04/04/20 alle ore 14:28:28