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Titolo:
Endotension: An explanation for continued AAA growth after successful endoluminal repair
Autore:
White, GH; May, J; Petrasek, P; Waugh, R; Stephen, M; Harris, J;
Indirizzi:
Univ Sydney, Royal Prince Alfred Hosp, Dept Vasc Surg, Sydney, NSW 2006, Australia Univ Sydney Sydney NSW Australia 2006 c Surg, Sydney, NSW 2006, Australia Univ Sydney, Royal Prince Alfred Hosp, Dept Intervent Radiol, Sydney, NSW 2006, Australia Univ Sydney Sydney NSW Australia 2006 Radiol, Sydney, NSW 2006, Australia
Titolo Testata:
JOURNAL OF ENDOVASCULAR SURGERY
fascicolo: 4, volume: 6, anno: 1999,
pagine: 308 - 315
SICI:
1074-6218(199911)6:4<308:EAEFCA>2.0.ZU;2-6
Fonte:
ISI
Lingua:
ENG
Soggetto:
ABDOMINAL AORTIC-ANEURYSMS; ENDOVASCULAR REPAIR; INTRAANEURYSMAL PRESSURE; 3-YEAR EXPERIENCE; GRAFT; ENDOLEAK; MANAGEMENT; EXCLUSION; RUPTURE; SAC;
Keywords:
abdominal aortic aneurysms; endovascular grafts; endoleak; intrasac pressure measurement; conversion;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Citazioni:
18
Recensione:
Indirizzi per estratti:
Indirizzo: White, GH Univ Sydney, Dept Surg, Endovasc Res Unit, Bldg D06, Sydney, NSW2006, Australia Univ Sydney Bldg D06 Sydney NSW Australia 2006 W 2006, Australia
Citazione:
G.H. White et al., "Endotension: An explanation for continued AAA growth after successful endoluminal repair", J ENDOVAS S, 6(4), 1999, pp. 308-315

Abstract

Purpose: To present and analyze several cases that illustrate persistent sac pressurization following endovascular abdominal aortic aneurysm (AAA) repair. Methods and Results: Four patients with successful endovascular AAA exclusion presented in follow-up with an expanding aneurysm. Two had initial sac diameter decrease, but by 18 and 24 months, respectively, the AAA had enlarged and become pulsatile. There was no endoleak evident, but the proximal attachment stents had migrated distally in both cases. One patient developedendoleak with aneurysm expansion at 6 months; contained rupture occurred at 12 months. The last case had slowly evolving aneurysm expansion over 36 months but no endoleak. All endografts were removed and successfully replaced with conventional grafts. Intrasac thrombus was implicated as the means of pressure transmission that precipitated AAA expansion in these cases. Conclusions: Excluded AAAs can increase in size owing to persistent or recurrent pressurization (endotension) of the sac even when there is no evidence of endoleak. One proposed mechanism is pressure transmission via thrombus that lines the attachment site. Endotension may also represent an indiscernible, very low flow endoleak that allows blood to clot at the source of leakage.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 01/12/20 alle ore 13:23:57