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Titolo:
Pain and sympathetic dysfunction in complex regional pain syndrome
Autore:
Birklein, F; Riedl, B; Griessinger, N; Neundorfer, B;
Indirizzi:
Univ Erlangen Nurnberg, Neurol Klin, D-91054 Erlangen, Germany Univ Erlangen Nurnberg Erlangen Germany D-91054 -91054 Erlangen, Germany Univ Erlangen Nurnberg, Anasthesiol Klin, D-91054 Erlangen, Germany Univ Erlangen Nurnberg Erlangen Germany D-91054 -91054 Erlangen, Germany
Titolo Testata:
NERVENARZT
fascicolo: 4, volume: 70, anno: 1999,
pagine: 335 - 341
SICI:
0028-2804(199904)70:4<335:PASDIC>2.0.ZU;2-N
Fonte:
ISI
Lingua:
GER
Soggetto:
DYSTROPHY; GUANETHIDINE; TEMPERATURE; SYMPTOMS;
Keywords:
complex regional pain syndrome; reflex sympathetic dystrophy; autonomic nervous system; clinical outcome;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Citazioni:
32
Recensione:
Indirizzi per estratti:
Indirizzo: Birklein, F Univermanygen Nurnberg, Neurol Klin, Schwabachanlage 6, D-91054 Erlangen, G Univ Erlangen Nurnberg Schwabachanlage 6 Erlangen Germany D-91054
Citazione:
F. Birklein et al., "Pain and sympathetic dysfunction in complex regional pain syndrome", NERVENARZT, 70(4), 1999, pp. 335-341

Abstract

In order to describe autonomic dysfunction and clinical outcome in complexregional pain syndrome (CRPS) 20 patients were followed-up. First investigation was performed in the acute stage of CRPS and the second investigationtwo years later after therapy (individual, not standardized). Skin temperature and sudomotor function (thermoregulatory sweating (TST) and quantitative sudomotor axon reflex (QSART)) were assessed, and a clinical follow-up was performed carefully. Skin temperature was warmer on the affected side atfirst investigation (p<0.001) and colder at follow-up (p<0.02). Sudomotor output was enhanced on the affected side both after TST (p<0.005) and QSART(p<0.05) at first investigation. At follow-up, however, while thermoregulatory swearing was still increased (p<0.04) QSART was not different. While autonomic failure improves as assessed by clinical examination, therapy failed to alleviate pain significantly. But patients' selfassesment of therapy was mostly positive (16 of 20,p<0.001). The present study has shown that the autonomic failure may be probably the result of central disturbances of thermoregulation, but secondary peripheral mechanisms also contribute to ourfindings. Individual based therapy seems to be efficacious for long term treatment,but for final judge controlled studies are required.

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Documento generato il 08/07/20 alle ore 06:55:31