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Titolo:
GENESIS OF ALZHEIMERS-DISEASE
Autore:
WHITEHOUSE PJ;
Indirizzi:
UNIV HOSP CLEVELAND,ALZHEIMER CTR,12200 FAIRHILL RD,SUITE C222 CLEVELAND OH 44120 CASE WESTERN RESERVE UNIV,DEPT NEUROL CLEVELAND OH 44106
Titolo Testata:
Neurology
fascicolo: 5, volume: 48, anno: 1997, supplemento:, 7
pagine: 2 - 7
SICI:
0028-3878(1997)48:5<2:GOA>2.0.ZU;2-E
Fonte:
ISI
Lingua:
ENG
Soggetto:
APOLIPOPROTEIN-E; CEREBROSPINAL-FLUID; PROTEIN; TAU; MECHANISMS; CHROMOSOME-14; ASSOCIATION; CYTOKINES; GENOTYPE; LOCUS;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
59
Recensione:
Indirizzi per estratti:
Citazione:
P.J. Whitehouse, "GENESIS OF ALZHEIMERS-DISEASE", Neurology, 48(5), 1997, pp. 2-7

Abstract

Our understanding of the pathogenesis of Alzheimer's disease (AD) hasadvanced rapidly, particularly in the area of genetics. Clinical trials of agents that offer the promise of going beyond symptomatic treatment to actually slowing the progression of disease, perhaps by enhancing the viability of neurons, are under way. Mast of the ideas for therapeutic approaches that may slow progression of disease have come froma combination of epidemiologic and basic science observations. Anti-inflammatory agents, nicotine, estrogens, and free radical scavengers are major areas of inquiry. In addition to being a biological conditionof considerable complexity, AD is also challenging from a social and cultural perspective. An appreciation of the changes occurring in our scientific and clinical environments may lead to building more effective bridges between science and society and to better diagnostic and therapeutic approaches to AD.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 28/09/20 alle ore 23:49:47