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Titolo:
TRANSIENT WEAKNESS AND COMPOUND MUSCLE ACTION-POTENTIAL DECREMENT IN MYOTONIA-CONGENITA
Autore:
DEYMEER F; CAKIRKAYA S; SERDAROGLU P; SCHLEITHOFF L; LEHMANNHORN F; RUDEL R; OZDEMIR C;
Indirizzi:
UNIV ISTANBUL,ISTANBUL FAC MED,DEPT NEUROL TR-34390 ISTANBUL TURKEY UNIV ULM,DEPT PHYSIOL D-89069 ULM GERMANY
Titolo Testata:
Muscle & nerve
fascicolo: 10, volume: 21, anno: 1998,
pagine: 1334 - 1337
SICI:
0148-639X(1998)21:10<1334:TWACMA>2.0.ZU;2-A
Fonte:
ISI
Lingua:
ENG
Soggetto:
REPETITIVE NERVE-STIMULATION; DIFFERENTIAL-DIAGNOSIS; SURFACE EMG; PARESIS;
Keywords:
MYOTONIA CONGENITA; RECESSIVE GENERALIZED MYOTONIA; TRANSIENT WEAKNESS; REPETITIVE NERVE STIMULATION; CHANNELOPATHIES;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
20
Recensione:
Indirizzi per estratti:
Citazione:
F. Deymeer et al., "TRANSIENT WEAKNESS AND COMPOUND MUSCLE ACTION-POTENTIAL DECREMENT IN MYOTONIA-CONGENITA", Muscle & nerve, 21(10), 1998, pp. 1334-1337

Abstract

Twenty-five Turkish patients with recessive myotonia congenita (RMC),16 of whom had genetic confirmation, were studied. Nineteen had transient weakness. In the upper extremities, onset age of transient weakness was usually in the early teens. All untreated RMC patients had a compound muscle action potential decrement of greater than or equal to 25%, usually above 50%, with repetitive nerve stimulation at 10/s for 5s. Patients with other nondystrophic diseases with myotonia, except 1patient with dominant myotonia congenita, had no transient weakness and a CMAP decrement below 25%. (C) 1998 John Wiley & Sons, Inc.

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Documento generato il 26/09/20 alle ore 11:55:14