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Titolo:
LEPTIN - AN INTERIM APPRAISAL
Autore:
BODNER J; EBENBICHLER CF; LECHLEITNER M; RITSCH A; SANDHOFER A; GANDER R; WOLF HJ; HUTER O; PATSCH JR;
Indirizzi:
INNSBRUCK UNIV,INNERE MED KLIN,ANICHSTR 35 A-6020 INNSBRUCK AUSTRIA INNSBRUCK UNIV,INNERE MED KLIN A-6020 INNSBRUCK AUSTRIA INNSBRUCK UNIV,KLIN GYNAKOL & GEBURTSHILFE A-6020 INNSBRUCK AUSTRIA
Titolo Testata:
Wiener Klinische Wochenschrift
fascicolo: 6, volume: 110, anno: 1998,
pagine: 212 - 219
SICI:
0043-5325(1998)110:6<212:>2.0.ZU;2-V
Fonte:
ISI
Lingua:
GER
Soggetto:
HUMAN OB GENE; MOUSE OBESE GENE; NEUROPEPTIDE-Y; PLASMA LEPTIN; ADIPOSE-TISSUE; MESSENGER-RNA; BODY-WEIGHT; OB/OB MICE; MISSENSE MUTATION; EXTREME OBESITY;
Tipo documento:
Review
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
95
Recensione:
Indirizzi per estratti:
Citazione:
J. Bodner et al., "LEPTIN - AN INTERIM APPRAISAL", Wiener Klinische Wochenschrift, 110(6), 1998, pp. 212-219

Abstract

The discovery of leptin, the product of the obese(ob)-gene, has broadened the horizons of research on energy balance. This hormone, produced and secreted by adipose tissue and some placental cells, finds its way to the hypothalamus, where it binds to the leptin receptors and signals satiety through the neuroendocrine axis. The fact that adipose tissue is not merely a storage depot, but also an important endocrine tissue, has revived the interest in the ''lipostatic'' theory of body fat regulation and has initiated many research efforts in the field of obesity, anorexia nervosa, bulimia, reproduction and haematology.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 28/03/20 alle ore 10:20:33