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Titolo:
Arterial baroreflex control during mild-to-moderate nitrous oxide narcosis
Autore:
Ostlund, A; Sundblad, P; Demetriades, AK; Linnarsson, D;
Indirizzi:
KarolinskaStockholm,t Physiol & Pharmacol, Sect Environm Physiol, SE-17177Karolinska Inst Stockholm Sweden SE-17177 ect Environm Physiol, SE-17177 Karolinska Hosp, Dept Anesthesiol & Intens Care, SE-17176 Stockholm, Sweden Karolinska Hosp Stockholm Sweden SE-17176 re, SE-17176 Stockholm, Sweden Univ London Univ Coll, Dept Physiol, Sch Med, London WC1E 6BT, England Univ London Univ Coll London England WC1E 6BT , London WC1E 6BT, England
Titolo Testata:
UNDERSEA & HYPERBARIC MEDICINE
fascicolo: 1, volume: 26, anno: 1999,
pagine: 15 - 20
SICI:
1066-2936(199921)26:1<15:ABCDMN>2.0.ZU;2-B
Fonte:
ISI
Lingua:
ENG
Soggetto:
PRESSURE; HUMANS; REFLEX;
Keywords:
carotid baroreceptors; heart rate; blood pressure; neck suction; nitrous oxide;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Life Sciences
Citazioni:
22
Recensione:
Indirizzi per estratti:
Indirizzo: Ostlund, A KarolinskaStockholm,t Physiol & Pharmacol, Sect Environm Physiol, SE-17177 Karolinska Inst Stockholm Sweden SE-17177 m Physiol, SE-17177
Citazione:
A. Ostlund et al., "Arterial baroreflex control during mild-to-moderate nitrous oxide narcosis", UNDERS HYP, 26(1), 1999, pp. 15-20

Abstract

Ostlund A, Sundblad P, Demetriades AK, Linnarsson D. Arterial baroreflex control during mild-to-moderate nitrous oxide narcosis. Undersea Hyper Med 1999; 26(1):15-20.-We hypothesized that light-to-moderate inert gas narcosismight play a role in bradycardia in divers by altering sensitivity or response dynamics of arterial baroreflexes. Carotid-cardiac and carotid-mean arterial pressure (MAP) baroreflex response curves were generated by applyingmultiple levels of neck pressure and suction. Seven healthy volunteers were studied during air breathing (control) and during inhalation of 39% nitrous oxide (N2O). Baseline (pre-stimulus) heart rate (HR) and MAP were not altered by N2O. Range, threshold level, saturation level, End delay of responses did not differ between conditions. For hypertensive stimuli, sensitivity of responses did not differ between air control and N2O inhalation, but for hypotensive stimuli, maximal response gain for HR tended to be reduced with N2O inhalation (P = 0.054). Our results speak against inert gas narcosis as a primary mechanism for hyperbaric bradycardia, but it remains possible that an attenuation of tachycardic responses to hypotensive stimuli playsa role.

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Documento generato il 13/07/20 alle ore 17:06:04