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Titolo:
Immunoblastic lymphadenopathy-like T cell lymphoma evolving into a massiveplasma cell proliferation with biclonal paraproteinemia
Autore:
Higuchi, T; Tada, J; Mori, H; Niikura, H; Omine, M; Kishimoto, K; Tate, G; Mitsuya, T;
Indirizzi:
Showanagawa Fujigaoka Hosp, Div Hematol Internal Med, Aoba Ku, Yokohama, Ka Showa Univ Yokohama Kanagawa Japan 2278501 nal Med, Aoba Ku, Yokohama, Ka Showa Univ, Fujigaoka Hosp, Div Pathol, Yokohama, Kanagawa 2278501, Japan Showa Univ Yokohama Kanagawa Japan 2278501 ohama, Kanagawa 2278501, Japan
Titolo Testata:
ACTA HAEMATOLOGICA
fascicolo: 3, volume: 100, anno: 1998,
pagine: 151 - 155
SICI:
0001-5792(1998)100:3<151:ILTCLE>2.0.ZU;2-7
Fonte:
ISI
Lingua:
ENG
Soggetto:
ANGIOIMMUNOBLASTIC LYMPHADENOPATHY; MALIGNANT-LYMPHOMA; BETA-CHAIN; EVOLUTION; REARRANGEMENTS; DYSPROTEINEMIA; RECEPTOR; LESIONS;
Keywords:
angioimmunoblastic lymphadenopathy; immunoblastic lymphadenopathy; paraproteinemia; plasma cell;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Clinical Medicine
Life Sciences
Citazioni:
27
Recensione:
Indirizzi per estratti:
Indirizzo: Higuchi, T Showaka,iv, Fujigaoka Hosp, Div Hematol Internal Med, Aoba Ku, 1-30 Fujigao Showa Univ 1-30 Fujigaoka Yokohama Kanagawa Japan 2278501 jigao
Citazione:
T. Higuchi et al., "Immunoblastic lymphadenopathy-like T cell lymphoma evolving into a massiveplasma cell proliferation with biclonal paraproteinemia", ACT HAEMAT, 100(3), 1998, pp. 151-155

Abstract

We present a case of immunoblastic lymphadenopathy-like T cell lymphoma (IBL-T) who subsequently developed a massive proliferation of plasma cells. At diagnosis of IBLT, the patient had polyclonal hypergammaglobulinemia and subsequently, while on chemotherapy, developed paraproteinemia with biclonal peaks and the IBLT lesion was replaced with a massive proliferation of CD38-positive plasma cells. The evolution was not likely to be attributed to a new neoplastic proliferation of B cells. It appeared that two B cell clones possibly had a growth advantage among the polyclonal B cells due to a depletion of suppressor T cells or to a disturbance in the immune system.

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Documento generato il 03/12/20 alle ore 21:14:39