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Titolo:
STUDY OF ETIOLOGIC FACTORS IN NEPHROLITHI ASIS - THE ROLE OF CRYSTALLIZATION INHIBITORS
Autore:
FARDELLA C; LASTRA M; ROJAS A; TORO M; PINOCHET C; ROSALES R; RETAMAL F; MELLADO Y; LOPEZ F; SH ES; HUETE I; MERY J; CAMPINO C; CAMPUSANO C; LOPEZ JM;
Indirizzi:
DEPT ENDOCRINOL,CASILLA 114-D SANTIAGO CHILE CATHOLIC UNIV CHILE,DEPT ENDOCRINOL METAB & NUTR SANTIAGO CHILE CATHOLIC UNIV CHILE,FAC MED,SERV CIRUG SANTIAGO CHILE
Titolo Testata:
Revista Medica de Chile
fascicolo: 8, volume: 122, anno: 1994,
pagine: 873 - 879
SICI:
0034-9887(1994)122:8<873:SOEFIN>2.0.ZU;2-O
Fonte:
ISI
Lingua:
SPA
Soggetto:
URINARY CITRATE EXCRETION; CITRIC-ACID COMPLEXES; POTASSIUM CITRATE; MAGNESIUM-OXIDE; AL(III) COMPLEXES; CRYSTAL-GROWTH; STONE DISEASE; CALCIUM; UROLITHIASIS; CALCIFICATION;
Keywords:
KIDNEY CALCULI; URINE; CITRATE; CRYSTALLIZATION;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
32
Recensione:
Indirizzi per estratti:
Citazione:
C. Fardella et al., "STUDY OF ETIOLOGIC FACTORS IN NEPHROLITHI ASIS - THE ROLE OF CRYSTALLIZATION INHIBITORS", Revista Medica de Chile, 122(8), 1994, pp. 873-879

Abstract

Background: five percent of consultations at the Emergency Room of Catholic University Hospital are due to nephrolithiasis. The causes of this high frequency remain unknown. Aim: to know the main metabolic andanatomic factors involved in the genesis of neprholithiasis. Patientsand methods: forty one patients (31 male) were studied presenting with a renal colic were studied as soon as the acute episode subsided andwithout diet modifications. Fasting blood calcium and creatinine and 24 h urine calcium, uric acid, citrate, magnesium and pH were measuredand an intravenous pyelogram was performed. Twenty one subjects without a history of nephrolithiasis were used as controls. Results: Patients with nephrolithiasis did not differ from controls in urinary calcium (159 +/- 67 and 172 +/- 67 mg/24 h respectively), uricosuria (417 +/- 171 and 431 +/- 121 mg/24 h respectively) or urinary magnesium (55 +/- 19 and 62 +/- 21 mg/24 h respectively, whereas urinary citrate was lower (219 +/- 172 vs 319 +/- 179 mg/24 h in controls p < 0.05). All patients had normal renal functions, urinary acidification and intravenous pyelogram. Seven percent of patients with nephrolithiasis had hypercalciuria, 2.4% had hyperuncosuria, 68.3% had a low urinary citrate and 44.4% had low urinary magnesium. Conclusions: in this sample, thereis a strong association of nephrolithiasis with low levels of crystallization inhibitors in special with urinary citrate, a crystallizationinhibitor.

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Documento generato il 27/11/20 alle ore 13:12:14