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Titolo:
SINGLE-FIBER ELECTROMYOGRAPHY IN NEUROMUSCULAR DISORDERS - CORRELATION OF MUSCLE HISTOCHEMISTRY, SINGLE-FIBER ELECTROMYOGRAPHY, AND CLINICAL FINDINGS
Autore:
BERTORINI TE; STALBERG E; YUSON CP; ENGEL WK;
Indirizzi:
UNIV TENNESSEE,DEPT NEUROL,855 MONROE AVE,ROOM 415 MEMPHIS TN 38163 MEMPHIS NEUROSCI CTR MEMPHIS TN 00000 UNIV HOSP UPPSALA UPPSALA SWEDEN FORSYTH MEM HOSP WINSTON SALEM NC 00000 UNIV SO CALIF,SCH MED LOS ANGELES CA 00000
Titolo Testata:
Muscle & nerve
fascicolo: 3, volume: 17, anno: 1994,
pagine: 345 - 353
SICI:
0148-639X(1994)17:3<345:SEIND->2.0.ZU;2-0
Fonte:
ISI
Lingua:
ENG
Soggetto:
CENTRAL CORE DISEASE;
Keywords:
ELECTROMYOGRAPHY; SINGLE FIBER; HISTOCHEMISTRY;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
37
Recensione:
Indirizzi per estratti:
Citazione:
T.E. Bertorini et al., "SINGLE-FIBER ELECTROMYOGRAPHY IN NEUROMUSCULAR DISORDERS - CORRELATION OF MUSCLE HISTOCHEMISTRY, SINGLE-FIBER ELECTROMYOGRAPHY, AND CLINICAL FINDINGS", Muscle & nerve, 17(3), 1994, pp. 345-353

Abstract

We performed single-fiber electromyography (SFEMG) and correlated theresults with muscle-biopsy histochemistry in 56 patients with variousneuromuscular diseases. Increased muscle-fiber density, delineated bySFEMG, was most prominent in diseases of ordinary denervation, namelymotor neuron disorders and peripheral neuropathies, and it correlatedwith histochemical fiber type grouping. Both phenomena reflect denervation followed by reinnervation. In patients with central core diseaseand rod disease, fiber density was not increased despite massive typeI fiber predominance. The normal distribution of type I fiber subtypes in those patients indicated that their fiber predominance was not due to sprouting and reinnervation, but probably to paucity of the type II fibers. In type I fiber hypotrophy with central nuclei, fiber density was increased, perhaps attributable to the small diameter and consequent denser packing of the type I fibers. Fiber density was slightly increased in the majority of patients with acid-maltase deficiency, limb-girdle dystrophy, and polymyositis, in nearly half with mitochondrial myopathy, and in 1 older Duchenne dystrophy patient. In these myopathic disorders, myogenous deinnervation (followed by reinnervation) isone possible explanation. Normal fiber density was present in all patients with muscle phosphorylase deficiency, myotonia congenita, and inthe hypokalemic periodic paralysis patients under age 40. (C) 1994 John Wiley and Sons, Inc.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 10/07/20 alle ore 14:04:57