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Titolo:
PONTINE VERSUS CAPSULAR PURE MOTOR HEMIPARESIS
Autore:
NIGHOGHOSSIAN N; RYVLIN P; TROUILLAS P; LAHAROTTE JC; FROMENT JC;
Indirizzi:
HOP NEUROL,DEPT NEUROL & CEREBROVASC DIS,SERV NEUROL,59 BD PINEL F-69394 LYON 3 FRANCE HOP NEUROL,DEPT NEURORADIOL F-69394 LYON 3 FRANCE
Titolo Testata:
Neurology
fascicolo: 11, volume: 43, anno: 1993,
pagine: 2197 - 2201
SICI:
0028-3878(1993)43:11<2197:PVCPMH>2.0.ZU;2-E
Fonte:
ISI
Lingua:
ENG
Soggetto:
LACUNAR INFARCTION; CEREBROVASCULAR-DISEASE; STROKE; OCCLUSION;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
33
Recensione:
Indirizzi per estratti:
Citazione:
N. Nighoghossian et al., "PONTINE VERSUS CAPSULAR PURE MOTOR HEMIPARESIS", Neurology, 43(11), 1993, pp. 2197-2201

Abstract

We prospectively studied 21 patients with pure motor hemiparesis (PMH). CT showed a capsular lesion consistent with the clinical syndrome in 15 patients (71%) and was repeatedly negative in the remaining six (29%). In all six patients with repeatedly negative CT, MRI showed a pontine paramedian infarct as the notable cause of PMH. Clinical findings could not definitely distinguish between capsular and pontine PMH, but the combination of dysarthria and a history of previous transient gait abnormality or vertigo favored a pontine location. Outcome at 3 months was characterized by persistent, moderate to severe disability in86% of patients with pontine PMH versus 46% in capsular PMH. Based onMRI and magnetic resonance angiographic findings, the presumed mechanism of pontine ischemic lesions was a lacunar process in most instances (86%).

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Documento generato il 24/11/20 alle ore 14:36:24