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Titolo:
VAGAL REGULATION OF ACID-SECRETION AND GASTRIN-RELEASE
Autore:
DEBAS HT; CARVAJAL SH;
Indirizzi:
UNIV CALIF SAN FRANCISCO,DEPT SURG,DEANS OFF,513 PARNASSUS AVE,S-224 SAN FRANCISCO CA 94143 UNIV CALIF SAN FRANCISCO,GASTROINTESTINAL RES LAB SAN FRANCISCO CA 94143
Titolo Testata:
The Yale journal of biology & medicine
fascicolo: 3-4, volume: 67, anno: 1994,
pagine: 145 - 151
SICI:
0044-0086(1994)67:3-4<145:VROAAG>2.0.ZU;2-W
Fonte:
ISI
Lingua:
ENG
Soggetto:
PERFUSED RAT STOMACH; SOMATOSTATIN SECRETION; VAGUS NERVE; STIMULATION; POLYPEPTIDE; HISTAMINE; BOMBESIN; HORMONE; INHIBITION; VAGOTOMY;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
25
Recensione:
Indirizzi per estratti:
Citazione:
H.T. Debas e S.H. Carvajal, "VAGAL REGULATION OF ACID-SECRETION AND GASTRIN-RELEASE", The Yale journal of biology & medicine, 67(3-4), 1994, pp. 145-151

Abstract

The vagus nerve plays a central role in the regulation of gastric acid secretion and gastrin release. The current understanding of the mechanisms involved in vagal regulation of acid secretion and gastrin release is reviewed. Thyrotropin-releasing hormone from the medullary raphe nuclei appears to be the central excitatory mediator of vagal actionin the dorsal motor nucleus. Vagal stimulation of the parietal cell occurs through M3 cholinergic receptors and via the release of histamine and gastrin from enterochromaffin-like cells and G-cells, respectively. Somatostatin exerts a tonic basal inhibition of both the parietal cell and the G-cell. Vagal stimulation suppresses somatostatin releasefrom delta cells, thereby ''disinhibiting'' these cells.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 24/11/20 alle ore 22:24:28