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Titolo:
POSTERIOR HYPOTHALAMIC CONTROL OF RABBIT TRACHEAL TENSION AND INVOLVEMENT OF CENTRAL HISTAMINERGIC NEURONS
Autore:
IWASE M; KANAMARU M; HOMMA I;
Indirizzi:
SHOWA UNIV,SCH MED,DEPT PHYSIOL 2,SHINAGAWA KU,1-5-8 HATANODAI TOKYO 142 JAPAN SHOWA UNIV,SCH MED,DEPT PHYSIOL 2,SHINAGAWA KU TOKYO 142 JAPAN
Titolo Testata:
Journal of the autonomic nervous system
fascicolo: 1, volume: 53, anno: 1995,
pagine: 69 - 76
SICI:
0165-1838(1995)53:1<69:PHCORT>2.0.ZU;2-W
Fonte:
ISI
Lingua:
ENG
Soggetto:
ROSTRAL VENTROLATERAL MEDULLA; CENTRAL NERVOUS-SYSTEM; RAT-BRAIN; STIMULATION; NASAL; PROJECTIONS; MECHANISM; RECEPTORS; AIRWAYS; REGION;
Keywords:
RABBIT; POSTERIOR HYPOTHALAMUS; ELECTRICAL STIMULATION; TRACHEAL TENSION; SYMPATHETIC NERVE; HISTAMINERGIC NEURON;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
33
Recensione:
Indirizzi per estratti:
Citazione:
M. Iwase et al., "POSTERIOR HYPOTHALAMIC CONTROL OF RABBIT TRACHEAL TENSION AND INVOLVEMENT OF CENTRAL HISTAMINERGIC NEURONS", Journal of the autonomic nervous system, 53(1), 1995, pp. 69-76

Abstract

The hypothalamus is involved in the control of the cardiovascular system, but airway tone is less well defined. In the posterior hypothalamus, histaminergic neuronal cell bodies are located. Effects of electrical stimulation in the posterior hypothalamus on tracheal tension and the cardiovascular system were examined in anesthetized, paralyzed andartificially ventilated rabbits. Tracheal tension was determined frompressure exerted on a balloon inserted in the trachea and measured bya pressure transducer. Electrical stimulation of the posterior hypothalamus caused tracheal tension to decrease, arterial blood pressure toincrease, and mild tachycardia followed by bradycardia. The tracheal tension decrease induced by posterior hypothalamic stimulation was notaffected by atropine nor by transection of either the superior laryngeal nerve or the vagus nerve, but was depressed by adrenoceptor blockade. Tracheal tension decrease was also reduced by pyrilamine, a histamine H-1-receptor antagonist, administered into the fourth ventricle, but was not affected by cimetidine, a histamine H-2-receptor antagonist. The stimulation sites where these effects were evoked were interspersed among the loci of histamine immunoreactive cell bodies previously reported. Results suggest that posterior hypothalamic neurons decreasetracheal tension through the sympathetic nervous system, and involve the histaminergic neurons in this route.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 27/11/20 alle ore 01:22:31