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Titolo:
DISSOCIATION BETWEEN FOOD-INTAKE, DIET COMPOSITION, AND METABOLISM INPARABIOTIC PARTNERS OF OBESE RATS
Autore:
HARRIS RBS; MARTIN RJ; BRUCH RC;
Indirizzi:
PENNINGTON BIOMED RES CTR,6400 PERKINS RD BATON ROUGE LA 70808 UNIV GEORGIA,DEPT FOODS & NUTR ATHENS GA 30602 LOUISIANA STATE UNIV,DEPT ZOOL & PHYSIOL BATON ROUGE LA 70803
Titolo Testata:
American journal of physiology. Regulatory, integrative and comparative physiology
fascicolo: 4, volume: 37, anno: 1995,
pagine: 874 - 883
SICI:
0363-6119(1995)37:4<874:DBFDCA>2.0.ZU;2-M
Fonte:
ISI
Lingua:
ENG
Soggetto:
LIPID-DEPLETING FACTOR; BODY-WEIGHT; FAT;
Keywords:
PARABIOSIS; BODY FAT; LIPOGENESIS;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
22
Recensione:
Indirizzi per estratti:
Citazione:
R.B.S. Harris et al., "DISSOCIATION BETWEEN FOOD-INTAKE, DIET COMPOSITION, AND METABOLISM INPARABIOTIC PARTNERS OF OBESE RATS", American journal of physiology. Regulatory, integrative and comparative physiology, 37(4), 1995, pp. 874-883

Abstract

When one member of a parabiosed pair of rats is overfed, its ad libitum-fed partner loses body fat in the absence of a statistically significant decrease in food intake. Three experiments investigated the relationships between food intake, metabolism, and body composition in this model. In vivo measurement of lipogenesis confirmed that loss of fatis associated with decreased fat deposition. When partners of overfedrats were compared with food-restricted single rats, proportional changes in body composition and metabolism were similar for the two treatments, although there was no significant change in the food intake of parabiotic rats, whereas restricted rats received only 60% of the intake of their controls. The final experiment demonstrated that changes in body composition of partners of overfed rats were independent of dietary composition. These results suggest that, when a rat is made obeseby overfeeding, a circulatory factor is released that inhibits fat deposition and disrupts regulatory mechanisms that normally stimulate food intake during a period of negative energy balance.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 19/09/20 alle ore 12:51:45