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Titolo:
FAMILIAL HYPERURICEMIC NEPHROPATHY
Autore:
REITER L; BROWN MA; EDMONDS J;
Indirizzi:
ST GEORGE HOSP,DEPT RENAL MED KOGARAH NSW 2217 AUSTRALIA ST GEORGE HOSP,DEPT RENAL MED KOGARAH NSW 2217 AUSTRALIA ST GEORGE HOSP,DEPT MED KOGARAH NSW 2217 AUSTRALIA ST GEORGE HOSP,DEPT RHEUMATOL KOGARAH NSW 2217 AUSTRALIA UNIV NEW S WALES SYDNEY NSW AUSTRALIA
Titolo Testata:
American journal of kidney diseases
fascicolo: 2, volume: 25, anno: 1995,
pagine: 235 - 241
SICI:
0272-6386(1995)25:2<235:FHN>2.0.ZU;2-W
Fonte:
ISI
Lingua:
ENG
Soggetto:
CHRONIC LEAD NEPHROPATHY; URIC-ACID; RENAL-DISEASE; GOUT;
Keywords:
FAMILIAR HYPERURICEMIA; PRECOCIOUS GOUT; URATE NEPHROPATHY; FRACTIONAL URATE EXCRETION; HYPERTENSION;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
24
Recensione:
Indirizzi per estratti:
Citazione:
L. Reiter et al., "FAMILIAL HYPERURICEMIC NEPHROPATHY", American journal of kidney diseases, 25(2), 1995, pp. 235-241

Abstract

This report describes a Polynesian family that had the rare combination of hyperuricemia, precocious gout, hypertension, and renal failure at an early age, with an autosomal dominant inheritance. One family member had renal biopsy evidence of interstitial urate crystal deposition, a surprisingly uncommon observation in such families, and most had decreased fractional excretion of urate, reflecting either decreased secretion or enhanced postsecretory renal reabsorption of uric acid. One patient has had a successful renal transplant. On the basis of theseobservations, family members of any such index case should be screened for this disorder. Treatment of affected members might include a uricosuric agent and/or allopurinol early in the course of the disorder. (C) 1995 by the National Kidney Foundation, Inc.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 21/09/20 alle ore 18:23:16