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Titolo:
MECHANISM OF BICARBONATE SECRETION IN RAT (RATTUS-RATTUS) COLON
Autore:
DAGHER PC; RHO JI; CHARNEY AN;
Indirizzi:
NYU,SCH MED,DEPT VET AFFAIRS MED CTR,NEPHROL SECT NEW YORK NY 10010 NYU,SCH MED,DEPT VET AFFAIRS MED CTR,NEPHROL SECT NEW YORK NY 10010
Titolo Testata:
Comparative biochemistry and physiology. Part A, Physiology
fascicolo: 1, volume: 105, anno: 1993,
pagine: 43 - 48
SICI:
1096-4940(1993)105:1<43:MOBSIR>2.0.ZU;2-2
Fonte:
ISI
Lingua:
ENG
Soggetto:
INTESTINAL ELECTROLYTE TRANSPORT; ACID-BASE-DISORDERS; ION-TRANSPORT; DISTAL COLON; BASOLATERAL MEMBRANE; CHLORIDE ABSORPTION; CARBONIC-ANHYDRASE; SODIUM; POLYPEPTIDE; COTRANSPORT;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Science Citation Index Expanded
Citazioni:
26
Recensione:
Indirizzi per estratti:
Citazione:
P.C. Dagher et al., "MECHANISM OF BICARBONATE SECRETION IN RAT (RATTUS-RATTUS) COLON", Comparative biochemistry and physiology. Part A, Physiology, 105(1), 1993, pp. 43-48

Abstract

1. Colonic HCO3 secretion was measured as the residual flux in male Sprague Dawley rats (Rattus rattus). 2. Basal HCO3 secretion was increased by 1 mM dibutyryl cyclic AMP (dbcAMP) and was reduced to baseline by 0.1 mM methazolamide (Mtz) but not by SITS (1 mM), DIDS (1 mM) or amiloride (1 mM). 3. In vivo, intravenous vasoactive intestinal peptideincreased HCO3 secretion and prior perfusion with 1 mM Mtz prevented this increase. 4. These results suggest that the source of basal and cAMP-stimulated HCO3 secretion is, in part, intracellular and requires the action of carbonic anhydrase.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 28/09/20 alle ore 15:41:23