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Titolo:
EPILEPTIC NEGATIVE MYOCLONUS - AN EEG-SINGLE-PHOTON EMISSION CT STUDYINDICATING INVOLVEMENT OF PREMOTOR CORTEX
Autore:
BAUMGARTNER C; PODREKA I; OLBRICH A; NOVAK K; SERLES W; AULL S; ALMER G; LURGER S; PIETRZYK U; PRAYER D; LINDINGER G;
Indirizzi:
UNIV VIENNA,NEUROL KLIN,WAHRINGER GURTEL 18-20 A-1090 VIENNA AUSTRIA KRANKENHAUS RUDOLFSTIFTUNG,NEUROL ABT VIENNA AUSTRIA MAX PLANCK INST NEUROL RES COLOGNE GERMANY UNIV VIENNA,KLIN RADIODIAGNOST VIENNA AUSTRIA
Titolo Testata:
Neurology
fascicolo: 3, volume: 46, anno: 1996,
pagine: 753 - 758
SICI:
0028-3878(1996)46:3<753:ENM-AE>2.0.ZU;2-Y
Fonte:
ISI
Lingua:
ENG
Soggetto:
STIMULATION; SEIZURES; ATONIA;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
25
Recensione:
Indirizzi per estratti:
Citazione:
C. Baumgartner et al., "EPILEPTIC NEGATIVE MYOCLONUS - AN EEG-SINGLE-PHOTON EMISSION CT STUDYINDICATING INVOLVEMENT OF PREMOTOR CORTEX", Neurology, 46(3), 1996, pp. 753-758

Abstract

We report a combined EEG-single-photon emission CT (SPECT) study on apatient with epileptic negative myoclonus (ENM). Clinically, the ENM was characterized by brief repetitive lapses in postural tone of the right upper extremity when the arms were held outstretched, whereas no movement effect was observed during rest. Ictal EEG showed repetitive left frontal spikes with a maximum at electrodes FC1 and Fl. EMG silent periods lasting from 100 to 200 ms followed the onset of the EEG transients by a latency of 20 to 40 ms. The N20 component of median nervesomatosensory evoked potentials-representing a biological marker of the location of central fissure-showed a phase reversal between electrodes P3 and C1 and thus was located considerably posterior to the spikemaximum. We obtained accurate anatomic reference of cerebral blood flow changes visible on SPECT by a special coregistration technique of MRI aid SPECT. SPECT performed during ENM showed a marked regional hyperperfusion in the left middle frontal gyrus aid a less pronounced increase in tracer uptake in the left supramarginal gyrus. Our results suggest that ENM is generated by epileptic activity in the premotor area in the middle frontal gyrus corresponding to Brodmann's area 6.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 17/09/19 alle ore 23:28:33