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Titolo:
ALTERATIONS IN MAP2 IMMUNOSTAINABILITY AFTER PROLONGED COMPLETE BRAINISCHEMIA IN THE RAT
Autore:
VANICKY I; BALCHEN T; DIEMER NH;
Indirizzi:
SLOVAK ACAD SCI,INST NEUROBIOL,SOLTESOVEJ 4 KOSICE 04001 SLOVAKIA UNIV COPENHAGEN,INST NEUROPATHOL,CEREBRAL ISCHEMIA RES GRP COPENHAGENDENMARK
Titolo Testata:
NeuroReport
fascicolo: 1, volume: 7, anno: 1995,
pagine: 161 - 164
SICI:
0959-4965(1995)7:1<161:AIMIAP>2.0.ZU;2-I
Fonte:
ISI
Lingua:
ENG
Soggetto:
MICROTUBULE-ASSOCIATED PROTEIN-2; COMPLETE CEREBRAL-ISCHEMIA; HIPPOCAMPUS; GERBILS; DAMAGE; CALPAIN;
Keywords:
BRAIN ISCHEMIA; CARDIAC ARREST; MAP2; IMMUNOCYTOCHEMISTRY; RAT;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
23
Recensione:
Indirizzi per estratti:
Citazione:
I. Vanicky et al., "ALTERATIONS IN MAP2 IMMUNOSTAINABILITY AFTER PROLONGED COMPLETE BRAINISCHEMIA IN THE RAT", NeuroReport, 7(1), 1995, pp. 161-164

Abstract

IMMEDIATE loss of MAP2 immunostainability after very short ischaemic periods (36 min) has been reported in the gerbil. With longer periods of ischaemia this loss of immunoreactivity becomes identical with the pattern of ischaemia-induced selective neuronal injury. In contrast, in the four-vessel occlusion rat model, no such changes were observed during the early recirculation period As no studies have focused on immediate changes due to complete brain ischaemia, we studied MAP2 immunoreactivity in the rat after cardiac arrest for up to 30 min. We observed a moderate decrease in stainability but the changes did not resemble those of selective vulnerability. We conclude that prolonged complete brain ischaemia without recirculation in the rat does not cause selective loss of MAP2 immunostainability in the vulnerable regions.

ASDD Area Sistemi Dipartimentali e Documentali, Università di Bologna, Catalogo delle riviste ed altri periodici
Documento generato il 04/07/20 alle ore 05:28:17