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Titolo:
2 MODES OF AUDITORY HEIR CELL LOSS FOLLOWING ACOUSTIC OVERSTIMULATIONIN THE AVIAN INNER-EAR
Autore:
NAKAGAWA T; YAMANE H; SHIBATA S; TAKAYAMA M; SUNAMI K; NAKAI Y;
Indirizzi:
YODOGAWA CHRISTIANS HOSP,DEPT OTORHINOLARYNGOL,2-9-26 AWAJI HIGASHIYODOGAWAKU OSAKA 533 JAPAN
Titolo Testata:
ORL
fascicolo: 6, volume: 59, anno: 1997,
pagine: 303 - 310
SICI:
0301-1569(1997)59:6<303:2MOAHC>2.0.ZU;2-8
Fonte:
ISI
Lingua:
ENG
Soggetto:
HAIR-CELLS; CHICK COCHLEA; NOISE DAMAGE; TRAUMA; REGENERATION; APOPTOSIS; DEATH; REORGANIZATION; ISCHEMIA;
Keywords:
ACOUSTIC TRAUMA; HAIR CELL; APOPTOSIS; CELL EXTRUSION; REPAIR;
Tipo documento:
Article
Natura:
Periodico
Settore Disciplinare:
Science Citation Index Expanded
Citazioni:
21
Recensione:
Indirizzi per estratti:
Citazione:
T. Nakagawa et al., "2 MODES OF AUDITORY HEIR CELL LOSS FOLLOWING ACOUSTIC OVERSTIMULATIONIN THE AVIAN INNER-EAR", ORL, 59(6), 1997, pp. 303-310

Abstract

To determine the type of cell death occurring and how the removal of damaged cells proceeds following overstimulation, we examined chick basilar papillae using an in situ DNA nick end labeling method and transmission electron microscopy. Two distinct modes of hair cell loss wereidentified. First, hair cells which had not progressed into typical cell death processes, apoptosis or ncecrosis, were deleted by extrusionfrom the epithelium just after sound exposure. Second, hair cells manifested degeneration through the process of apoptosis, then further deterioration within the epithelium after the beginning of the process of hair cell regeneration. The latter mode may contribute to the following repair processes.

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Documento generato il 02/12/20 alle ore 08:13:10